Release date: 2016-06-12
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-- Recently, researchers from the US Department of Energy's Lawrence Berkeley National Laboratory and the University of California found that copper may play an important role in fat metabolism; related research published in the international journal Nature Chemical Biology .
Because of its malleability and conductivity, copper plays an important role in the cooking tools, electronics, and lead industries. In recent years, scientists have been paying more and more attention to the role of copper in the specific biological functions of the body. The important role that scientists now know is that copper can help form red blood cells, absorb iron ions, promote connective tissue development, and the body's immune system.
Researcher Chang said that we found that copper can help metabolize fat cells so that fat metabolites can be used by the body as energy; therefore, copper plays a role as a regulator. The higher the level of copper, the higher the level of fat metabolism. Moreover, researchers believe that it is very necessary to study whether the loss of nutrients is directly related to the risk of obesity and obesity-related diseases.
The researchers pointed out that copper plays an important role in restoring the body's natural ways of burning fat. Copper is abundant in food, such as oysters and other shellfish, green leafy vegetables, mushrooms, seeds, Nuts and beans. According to data from the Food and Nutrition Board, the average daily copper requirement for an adult is about 700 mg, while in Rose, only 25% of adult individuals achieve daily copper intake.
The researchers used a special genetically mutated mouse that could not accumulate copper in the liver due to genetic mutations. The researchers used this to study the association between copper and fat. It is worth noting that these mice The average fat accumulation in the liver is higher than that in normal mice; for example, a genetic disease called Wilson's disease that causes excessive copper ions in the body and causes death.
The researchers conducted a study of mice with Wilson's disease and found that abnormal accumulation of copper in the body of diseased mice is often accompanied by lower fat levels in the liver of mice compared to control mice. The researchers also pointed out that white adipose tissue in the body of diseased mice usually contains lower levels of copper. The researchers then used Wilson's disease agonist, isoproterenol, which induces lipid degradation. The lipid degradation process is the process of breaking fat into fatty acids, often through the cyclic adenosine signaling pathway (cAMP). To complete, the researchers found that the diseased mice showed lower fat-degrading activity than the control.
Subsequently, the researchers conducted cell culture experiments to reveal the mechanism of copper affecting lipid degradation. They used Inductively Coupled Plasma Mass Spectrometry to determine the level of copper in adipose tissue. It was found that copper can be combined with a kind of copper. An enzyme of phosphodiesterase 3 (PDE3), which binds to cAMP, thereby blocking the ability of cAMP to promote fat breakdown.
Researchers believe that the association between copper and fat metabolism is very exciting, and the level of copper in the brain is high. Recently, many studies have found that copper can help brain cells communicate by acting as a brake. The original study by the researcher Chang is to clarify the role of copper in the communication between nerve cells. In this study, he studied the important role of copper in fat metabolism and other biological pathways.
Source: Bio Valley
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